SCI论文投稿coverletter

论文投稿时一般要求附带一篇“CoverLetter”，好的“CoverLetter”不仅可以使文章锦上添花，更有可能直接决定论文是否被录用。本人切身体会，以已发表论文为例，现将其写法经验归纳如下：DearEditor,Wewouldliketosubmittheenclosedmanuscriptentitled论文题目,whichwewishtobeconsideredforpublicationin期刊名.Theworkdescribedhasnotbeensubmittedelsewhereforpublication,inwholeorinpart,andalltheauthorslistedhaveapprovedthemanuscriptthatisenclosed.Webelievethattwoaspectsofthismanuscriptwillmakeitinterestingtogeneralreadersofyourjournal.First,论文亮点1.Second,论文亮点2.Thankyouverymuchforyourtimeandconsideration.Sincerelyyours,作者名注意点：论文亮点一定要简洁明了，一定要有创新，突出你的文章与其它已发表论文的区别。这是CoverLetter的主要内容。1、什么是coverletter?指的是SCI论文投稿信2、coverletter的内容主要包括那些?应该简述所投稿件的核心内容、主要发现和意义,拟投期刊,对稿件处理有无特殊要求等(如“nottoreview”list)。另外,请附上主要作者的中文姓名、通讯地址、电话、传真和e-mail地址。此外有的杂志要求推荐几位审稿人及其联系方式。以及谁已经阅读过该文(当然是牛人)。SCI论文发表投稿信写作相关知识，请咨询“辑文编译”。我投的那个杂志是要求说明你论文研究的意义,以及与这个杂志的相关性,另外还有的可能要写明你没有一搞多投等。此外临床实验要求写明符合伦理学要求。3、如何写SCI论文coverletter?各个杂志的具体要求是不一样的,在杂志的guideforauthors一般会有要求。如果没有具体的要求,大家可按照通用要求处理。SCI论文发表投稿信写作相关知识，请咨询“辑文编译”。4、SCI论文投稿信常用模板:(1)DearMr.**1.Theworkdescribedhasnotbeensubmittedelsewhereforpublication,inwholeorinpart,andalltheauthorslistedhaveapprovedthemanuscriptthatisenclosed.2.IhavereadandhaveabidedbythestatementofethicalstandardsformanuscriptssubmittedtoNeuroscience.kindregards.Yoursincerely,通讯作者(2)DearDr.主编name:Wesubmitourmanuscriptentitled文章titleto杂志名forpublication.接着简单介绍你文章的主要创新点和意义,不易过多,但要突出新意和关键点。Allauthorshaveseenthemanuscriptandapprovedtosubmittoyourjournal.Thankyouverymuchforyourattentionandconsideration.Sincerelyyours,通讯作者(3)DearDr.主编name:Wesubmitourmanuscriptentitled文章titleto杂志名forpublication.接着简单介绍你文章的主要创新点和意义,不易过多,但要突出新意和关键点。Allauthorshaveseenthemanuscriptandapprovedtosubmittoyourjournal.Thankyouverymuchforyourattentionandconsideration.Sincerelyyours,通讯作者(4)Dr.***Editor-in-Chief,***(addaddress)January22,2003DearDr.**,Enclosedherewithpleasefind3copiesofaMSby:‘***.***and***'entitled:“**********”,whichwewouldliketosubmitforpublicationinthe‘******'.Lookingforwardtoyourdecision,Withkindpersonalregards,Sincerelyyours,*****Professorof***(5)DearProf.***:Thisisamanuscriptby**and**entitled“……”。Itissubmittedtobeconsideredforpublicationasa“…”inyourjournal.Thispaperisnew.Neithertheentirepapernoranypartofitscontenthasbeenpublishedorhasbeenacceptedelsewhere.Itisnotbeingsubmittedtoanyotherjournal.Webelievethepapermaybeofparticularinteresttothereadersofyourjournalasit……Correspondenceshouldbeaddressedto**atthefollowingaddress,phoneandfaxnumber,andemailaddress:…Thanksverymuchforyourattentiontoourpaper.Sincerelyyours,CI论文投稿coverletter投稿信模版DeareditorialboardofEuropeanJournalofCardiology,Pleasefindenclosedthemanuscript:“Theangiotensin-convertingenzymeisnotariskfactorformyocardialinfarctioninFrenchindividuals”,bySarahH.,etal.,tobesubmittedasaShortCommunicationtotheEuropeanJournalofNeurologyforconsiderationofpublication.Allco-authorshaveseenandagreewiththecontentsofthemanuscriptandthereisnofinancialinteresttoreport.Wecertifythatthesubmissionisoriginalworkandisnotunderreviewatanyotherpublication.Inthismanuscript,wereporttheresultsofthefirststudyonthegeneticandfunctionalrolesoftheACEontheriskofsufferingamyocardialinfarctionintheFrenchpopulation.Indeed,wegenotypedthers4341polymorphismin531IScasesand549healthycontrols,andthenperformedfunctionalstudiesbymeasuringserumACEproteinlevelandactivityinhealthycontrols,strokepatientsatbaselineandstrokepatients24hafterstrokesymptomsonset.TheresultsfromourstudydidnotrevealanyassociationoftheACEvariantwithmyocardialinfarction,althoughitaffectedACEproteinlevel,andischemicstrokepatientsshowedlowerACElevelthancontrolsintheacutephasebutnotinthechronicphase.WebelievethatourfindingscouldbeofinteresttothereadersofEuropeanJournalofCardiologybecausetheybringnewandstrongevidencethattheACEgeneandproteinarenotariskfactorformyocardialinfarction.Wehopethattheeditorialboardwillagreeontheinterestofthisstudy.Sincerelyyours,SarahH.onbehalfoftheauthors.Correspondingauthor:SarahHamiltonatCardiovascularResearchLaboratory,MarieCurieResearchInstitute,75000,Paris,France,[e-mail],phonenumber:[phone]xxx,faxnumber:[phone]xxx.Case1DearEditor,WewouldliketosubmittheenclosedmanuscriptentitledGDNFAcutelyModulatesNeuronalExcitabilityandA-typePotassiumChannelsinMidbrainDopaminergicNeurons,whichwewishtobeconsideredforpublicationinNatureNeuroscience.GDNFhaslongbeenthoughttobeapotentneurotrophicfactorforthesurvivalofmidbraindopaminergicneurons,whicharedegeneratedinParkinson’sdisease.Inthispaper,wereportanunexpected,acuteeffectofGDNFonA-typepotassiumchannels,leadingtoapotentiationofneuronalexcitability,inthedopaminergicneuronsincultureaswellasinadultbrainslices.Further,weshowthatGDNFregulatestheK+channelsthroughamechanismthatinvolvesactivationofMAPkinase.Thus,thisstudyhasrevealed,forthefirsttime,anacutemodulationofionchannelsbyGDNF.OurfindingschallengetheclassicviewofGDNFasalong-termsurvivalfactorformidbraindopaminergicneurons,andsuggestthatthenormalfunctionofGDNFistoregulateneuronalexcitability,andconsequentlydopaminerelease.Theseresultsmayalsohaveimplicatio